[1]
Radwańska-Wala B., Buszman E, Drużba D.: Udział reaktywnych form
tlenu w patogenezie chorób ośrodkowego układu nerwowego. Wiad.
Lek. 2008; LXI: 1-3.
[2]
Gruber B.: Enzymy chroniące organizm przed reaktywnymi postaciami
tlenu. Farm Pol 1996; 52: 263–271.
[3]
Delanty N., Dichter M. A.: Oxidative injury in the nervous system. Acta
Neurol. Scand., 1998; 98: 145-153.
[4]
Montine J.T., Neely M.D., Quinn F., et al.: Lipid peroxidation in aging
brain and Alzheimer’s disease. Free Radic Biol Med 2002; 33: 620–626.
[5]
Sołowiej E., Sobaniec W.: Ocena wpływu CBZ i VPA na aktywność
enzymów antyoksydacyjnych oraz procesy lipidowej peroksydacji w
surowicy młodych pacjentów z padaczką. Neurol. Neurochir. Pol. 2003;
T.37(LIII), NR 5: 991-1003.
[6]
Bartosz G.: Druga twarz tlenu. Wolne rodniki w przyrodzie. Wyd. Nauk.
PWN, Warszawa 2003.
[7]
Ordonez F., Rosety-Plaza M., Rosety-Rodriguez M.: Glucose-6-phosphatedehydrogenase
is also increased in erythrocytes from adolescents with
Down syndrome. Down Syndrome Research and Practice 2006; 11(2):
84-87.
[8]
Tan W., Yeung E.S.: Simultaneous determination of enzyme activity and
enzyme quantity in single human erythrocytes. Anal. Biochem. 1995;
226: 74-79.
[9]
Richards R.S., Roberts T.K., Dunstan R.H., et al.: Erythrocyte antioxidant
systems protect cultured endothelial cells against oxidant damage.
Biochem Mol Biol Int 1998; 46: 857-865.
[10]
Tsukahara H., Jiang M.Z., Ohta N.: Oxidative stress in neonates:
evaluation using specific biomarkers. Life Sci 2004; 75: 933–938.
[11]
Śmigielska –Kuzia J., Sobaniec W., Kułak W., et al.: Antioxidant
enzymes and lipid peroxides in children with Down syndrome. J Pediatr
Neurol 2007; 5: 117-120.
[12]
Sobaniec H., Sobaniec W., Sendrowki K., et al.: Antioxidant activity of
blood serum and saliva in patients with periodontal disease treated due
to epilepsy. Adv Med Sci, 2007; 52(1): 204-206.
[13]
Artemowicz B., Sobaniec W.: Badanie procesów peroksydacji lipidów
oraz aktywności enzymów antyoksydacyjnych w surowicy dzieci z
padaczką. Epileptologia, 2002, 9:167-178.
[14]
Sobaniec W., Sołowiej E., Kułak W., et al.: Evaluation of the influence
of antiepileptic therapy on antioxidant enzyme activity and lipid
peroxidation in erythrocythes of children with epilepsy. J Child Neurol
2006; 21(7): 558-562.
[15]
Kułak W., Sobaniec W., Sołowiej E., et al.: Antioxidant enzymes and lipid
peroxides in children with cerebral palsy. Life Sci 2005; 77: 3031-3036.
[16]
Boćkowski L., Sobaniec W., Kułak W., et al.: Serum and intraerythocyte
enzymes and lipid peroxides in children with migraine. Pharmacol Rep.
2008; 60: 542-548.
[17]
Boćkowski L., Sobaniec W., et al.: Aktywność układów
antyoksydacyjnych i procesów peroksydacji lipidów u dzieci z
migrenowymi bólami głowy. Neurol Dziec 2007; 16: 34-35.
[18]
Sykes J. A., McCormack F.X., O’Brien T.J.: A preliminary study of
superoxide dismutase content of some human tumors. Cancer Res.
1978; 38: 2759-2762.
[19]
Paglia D.E., Valentine W. N.: Studia on quantative and qualitive
characterization of erythrocyte glutathione peroxidase. J. Lab. Clin.
Med. 1967: 70158-169.
[20]
Mize C.E., Landgon R.G.: Hepatic glutathione reductase I. Purification
and general kinetic properties. Biol. Chem. 1962; 237: 1589-95.
[21]
Buege J.A., Aust S.D.: Microsomal lipid peroxidation. Methods Enzymol.
1978; 51: 302-310.
[22]
Thiel S., Fowkes S.W.: Can cognitive deterioration associated with
Down syndrome be reduced?. Med Hypotheses 2995; 64 (3): 524-32.
[23]
Muchova J., Garaiova I., Sustrova M.: The redox state of glutathione
in erythrocytes of individuals with Down syndrome. Bratisl Lek Listy.
2007; 108(2):70-4.
[24]
Pastor M.C., Sierra C., Doladé M., et al.: Antioxidant enzymes and fatty
acid status in erythrocytes of Down’s syndrome patients. Clin Chem
1998; 44: 924–929.
[25]
Habif S., Mutaf I., Turgan N., et al.: Age and gender dependent alterations
in the activities of glutathione related enzymes in healthy subjects. Clin
Biochem 2001; 34: 667-671.
[26]
Andersen H.R., Nielsen J.B., Nielsen F.: Antioxidative enzyme activities
in human erythrocytes. Clin Chem 1997; 43: 562-568.
[27]
Guemouri L., Artur Y., Herbeth B., et al.: Biological variability of
superoxide dismutase, glutathione peroxidase, and catalase in blood.
Clin Chem 1991; 37: 932-1937.
[28]
Gerli G., Zenoni L., Locatelli G.F., et al.: Erythrocyte antioxidant system in
Down syndrome. Am J Med Genet 1990; 7: 272-273.
[29]
Gracez M.E., Peres W. and Salvador M.: Oxidative stress and
hematologic and biochemical parameters in individuals with Down
syndrome. Mayo Clin Proc 2005; 80: 1607-1611.
[30]
Percy M.E., Dalton A.J., Markovic V.D., et al.: Red cell superoxide
dismutase, glutathione peroxidase and catalase in Down syndrome
patients with and without manifestations of Alzheimer disease. Am J
Med Genet 1990; 35: 459-467.
[31]
Pastore A., Tozzi G., Gaeta L.M., et al.: Glutathione metabolism and
antioxidant enzymes in children with Down syndrome. J Pediatr 2003;
142 (5): 583-5.
[32]
Muchova J., Sustrova M., Garaiova I., et al.: Influence of age on activities
of antioxidant enzymes and lipid peroxidation products in erythrocytes
and neutrophils of Down syndrome patients. Free Radic Biol Med 2001
Aug 15; 31(4): 499-508.
[33]
Meguid Nagwa A.,Kholoussi N.M., Afifi H.H.: Evaluation of superoxide
dismutase and glutathione peroxidase enzymes and their cofactors in
Egyptian children with Down’s syndrome. Biol. Trace Elem. Res. 2001;
1 (81): 21-8.
[34]
Brooksbank B.W., Balazs R.: Superoxide dismutase, glutathione
peroxidase and lipidperoxidation in Down’s syndrome fetal brain. Brain
Res 1984; 318(1): 37-44.
[35]
Busciglio J., Yanker B.A.: Apoptosis and increased generation of
reactive oxygen species in Down’s syndrome neurons in vitro. Nature
1995; 378(6559): 776-9.
[36]
Carratelli M., Porcaro L., Ruscica M., et al.: Reactive oxygen metabolites
and prooxidant status in children with Down’s syndrome. Int J Clin
Pharmacol Res 2001; 21 (2): 79-84.
[37]
de Haan J.B., Susil B., Pritchard M., et al.: An altered antioxidant balance
occurs in Down syndrome fetal organs: implications for the „gene
dosage effect” hypothesis. J Neural Transm Suppl 2003; 67: 67-83.
[38]
Gromadzinska J, Wasowicz W, Sklodowska M, et al.: Glutathione
peroxidase activity, lipid peroxides and selenium status in blood in
patients with Down’s syndrome. J Clin Chem Clin Biochem 1988; 26(5):
255-8.
[39]
Jovanovic S.V., Clements D., MacLeod K.: Biomarkers of oxidative
stress are significantly elevated in Down syndrome. Free Radic Biol Med
1998; Vol. 25, No 9: 1044-1048.
[40]
Zitnanova I., Korytar P., Sobotova H., et al.: Markers of oxidative stress in
children with Down syndrome. Clin Chem Lab Med 2006; 44: 306–310.